New research from Edith Cowan University (ECU) suggests that even a single session of targeted exercise could play a role in combating cancer. Francesco Bettariga, a PhD student at ECU, discovered that both resistance training and high-intensity interval training (HIIT) trigger the release of myokines—muscle-produced proteins with anti-cancer properties. His findings indicated that a single exercise session could reduce cancer cell proliferation by 20 to 30 percent.
Bettariga’s study involved breast cancer survivors, with myokine levels measured before, immediately after, and thirty minutes following one bout of either resistance training or HIIT. Both exercise approaches showed an increase in myokine levels, suggesting that even brief activity can activate protective cellular mechanisms against cancer.
Exercise, Inflammation, and Cancer Recurrence
According to a Science Daily report, Bettariga’s research also explored the impact of regular exercise on body composition and inflammation—a key factor in breast cancer recurrence and mortality. Persistent inflammation can encourage tumor progression by affecting cell proliferation, survival, invasiveness, and metastasis, while simultaneously suppressing immune system function. Because cancer itself and treatment side effects can raise inflammatory markers, breast cancer survivors often face a higher risk of recurrence and disease progression.
Understanding the Link Between Inflammation and Cancer
An article from the Cancer Center highlights that inflammation is now recognized as a hallmark of cancer, with estimates suggesting that it contributes to up to 20 percent of cancer-related deaths. Chronic or improperly regulated inflammation can harm healthy tissues over time, leading to DNA mutations and rapid cell reproduction that creates a favorable environment for tumor growth.
Recent studies indicate that persistent inflammation may encourage cancer in two main ways: by creating conditions that facilitate tumor formation and by supporting the growth and spread of existing cancer. The inflammatory process produces cytokines, molecules that promote the formation of new blood vessels to nourish tumors. It also generates free radicals, which can further damage DNA and drive malignant transformation.
Bettariga’s research underscores the potential of structured exercise as a non-pharmacological strategy to reduce inflammation and promote anti-cancer effects, highlighting the importance of physical activity for breast cancer survivors and offering a promising avenue for future therapeutic interventions.
Bettariga’s study involved breast cancer survivors, with myokine levels measured before, immediately after, and thirty minutes following one bout of either resistance training or HIIT. Both exercise approaches showed an increase in myokine levels, suggesting that even brief activity can activate protective cellular mechanisms against cancer.
Exercise, Inflammation, and Cancer Recurrence
According to a Science Daily report, Bettariga’s research also explored the impact of regular exercise on body composition and inflammation—a key factor in breast cancer recurrence and mortality. Persistent inflammation can encourage tumor progression by affecting cell proliferation, survival, invasiveness, and metastasis, while simultaneously suppressing immune system function. Because cancer itself and treatment side effects can raise inflammatory markers, breast cancer survivors often face a higher risk of recurrence and disease progression.
Understanding the Link Between Inflammation and Cancer
An article from the Cancer Center highlights that inflammation is now recognized as a hallmark of cancer, with estimates suggesting that it contributes to up to 20 percent of cancer-related deaths. Chronic or improperly regulated inflammation can harm healthy tissues over time, leading to DNA mutations and rapid cell reproduction that creates a favorable environment for tumor growth.
Recent studies indicate that persistent inflammation may encourage cancer in two main ways: by creating conditions that facilitate tumor formation and by supporting the growth and spread of existing cancer. The inflammatory process produces cytokines, molecules that promote the formation of new blood vessels to nourish tumors. It also generates free radicals, which can further damage DNA and drive malignant transformation.
Bettariga’s research underscores the potential of structured exercise as a non-pharmacological strategy to reduce inflammation and promote anti-cancer effects, highlighting the importance of physical activity for breast cancer survivors and offering a promising avenue for future therapeutic interventions.
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